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Atypical Odontalgia (AO) is a persistent pain condition located in the teeth and jaws. It has been described as a persistent neuropathic pain that. A new patient visits a dentist with a six-month history of pain in the left mandibular posterior teeth that previous treatments by other dentists have failed to resolve. Abstract. Objective. Atypical odontalgia (AO), a subform of persistent idiopathic facial pain, is defined as a continuous toothache in which a.

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Trang Thi Huyen Tu, Email: The International Classification of Headache Disorders, 3rd edition beta version.

Pre-Trigeminal Neuralgia Similar to Atypical Odontalgia: A Case Report

In our cases, AO might have developed regardless of whether or not the patients had a psychiatric history. His pain at night resolved within a few days, but remained in the daytime.

Symptoms of PTN are similar to and difficult to differentiate from those of dental disease [ 2 – 8 ]; thus, some patients with PTN undergo unnecessary dental treatment [ 245 ]. The investigation of psychopathology on this scale is different from other psychopathological scales used in this study. On the basis of these results, we suggest that a biopsychosocial model can be used to predict AO.

A case-control study was used to compare AO patients with control groups that presented with other forms of facial pain and with a pain-free control group PF. Furthermore, in case 2, the symptoms occurred in a clinical situation that did not involve dental treatment, so the onset of AO seems to vary among individuals.


In the first case, a year-old woman presented with a heavy, splitting pain in the four maxillary front post-crown teeth, as if they were being pressed from the side. Five months before the initial visit, she had visited a dental clinic after a metal inlay on her left mandibular first molar had detached.

Patients atypicwl to each BDHI item using a true or false format. She told us that she had strong anxiety for a pain appearance. Combination therapy of atypical odontalgia with fluoxetine and clonazepam: Time table of medicines prescription.

Investigating the difference in pharmacotherapeutic responses might help to advance the treatment of AO. The patient strongly requested a prescription of 7. Only 96 were approached personally, and, using the previous criteria, just a total of 65 subjects were selected check the flow chart.

Biopsychosocial Aspects of Atypical Odontalgia

Low-dose Aripiprazole augmentation in Amitriptyline-resistant burning mouth syndrome: May 29, ; Published date: This technique is a semiquantitative method used to quantify sensory nerve dysfunctions in patients with neuropathic pain [ 19 — 21 ]. Psychosomatic problems in dentistry. The interview included demographic, family, and social data as well as any distressing events that the individuals had experienced in the last 6 months.

She was unable to do her housework and tended to to lie down because of her pain. The dominant opinion is that BoNTA acts by inhibiting the exocytosis of local nociceptive neuropeptides, such as substance P and calcitonin gene-related peptide CGRPand excitatory neurotransmitters such as glutamate.


Clinical features of atypical odontalgia; three cases and literature reviews

International Headache Society criteria for trigeminal Neuralgia. Panoramic view shows site of the extracted tooth Click here to view. The patient also underwent root canal treatment to the left mandibular first molar at this time, but again there was no improvement in his pain. The only exception was somatization, which was higher in the TMD group. The aim of the study is to explore the presence of specific abnormalities in facial pain patients that can be considered as psychophysical factors predisposing to AO.

A statistical consideration of 65 cases. Current evidence on atypical odontalgia: Depression and resentment were positively correlated in all pain subjects Spearman rho coefficient 0.

Her psychiatric history revealed a panic disorder and her brother had committed suicide because of depression. A diagnosis of TN cannot be made until the development of a paroxysmal pattern of pain [ 19 ]. The high number of pain comorbid syndrome and the narrowness of the inclusion criteria allowed us to include only 65 of the subjects with facial pain initially selected.

The structure is made up of three factors or classes Sensorial, Affective, and Evaluative. Diagnosis and clinical management.